: Archive of SID. LDL t. Apo B Apo B-100 : LDL LDL HDL LDL LDL. . IRCT N1 *

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Ἰεζάβελ Γούναρης
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1 B * 1 1 F.esfarjani@yahoo.com : : :. * 91/10/12 : [4] d-.[1]. B/E %28 90/8/9 : 2 4.[1 2] B-100 : /4 29 : 8. (N = 15) (N =14) mg/dl. % B-100 t.. α<0/05 8 B100 :.(p<0/05). : B B-100 :.[3].[1]. IRCT N1 *

2 %43 %19. A-I.[1].[6] B-100 V B-100 [2] B-100.[6] V.[6] B-100.[2].[6]..[1].[5].[7 8]..[8] %1.[5].[1].[1 5]. -C..[1].[1].[1].[1 3 4]

3 : B TG TC B. (ROCSH). %18 %1 %20.[9]. 1 CE-120. = 0 / ( 4) 0 / ( 4 2 ) + 0 /02963 (Age) + 1 / 4072 : 4 %37 0/6.[5 10].[ ] (%40-60) (%15-20).[11].[11 12] B- 100 B- [6] 100 RCT mg/dl 34 1 Randomized Controlled Trials

4 t -..[13] VO2max ( ml /kg/min)=132/ 85-0/ 0769(bodyweight) - 0/3877(age) + 6/ 315(sex) - 3/ 2649(time) - 0/1565(heart rate) 16 SPSS α<0/ BMI (Kg) (cm) ( ) 30 /6 76 / / 9 44 / 2 n=14 30 /5 75 / / 1 42 / 7 n=15 t -. B100.(2). 8 0/005 0/272 0/03 0/401 0/619 0/ : % [ ] F 9/36 1/25 2/47 0/72 0/25 4/ / / /31 377/37 16/ /87 ( ) (mg/dl) (mg/dl) (mg/dl) (mg/dl) (mg/dl) B100 (mg/dl)

5 %30 28.[14].[17]. GLUT4.[18]. GLUT-4 [8].[8 16] GLUT4 *0/002 0/77 *0/02 0/57 *0/02 0/14 0/86 0/23 *0/04 0/75 0/46 0/38 0/43 0/85 *0/03 0/71.[8 5] GLUT4 8 B100.(p<0/05) 8. %9/2.(3 ) 8 (FBS) mg/dl 8 (%16/2) 155/2 185/ %20 (2009) 2 %18/2 (2009) [16] % T 3/722 0/253 2/67 0/65 2/82 1/12 0/180 1/18 1/96 0/270 0/88 1/23 0/96 0/165 2/95 0/86 30/2±5/6 29/9±5/1 29/1±4/1 25/8±4/76 155/2±55/4 204/4±78/2 197/35±52/1 202/8 ±40/6 193/2±46/4 204/4± 76/6 111/7±46/5 115/4±32/7 48/1±6/6 42/3±8/1 94±10/1 95±9/4 31/3±5/3 29/3±4/9 26/6±5/5 24/3±4/1 185/8±53/2 190/7±69/4 199/2±35/3 188/6 ±33/6 212 ±99/1 200/4± 68/6 113/8±31/7 109/9±29/5 47±11/8 43/80±4/1 98±11/9 96±8/9 10 (%) (ml.kg- 1.min -1 ) ( mg/dl) (mg/dl) (mg/dl) (mg/dl) (mg/dl) (mg/dl) B100 * 1 Arara 2 Bweir

6 [19].[20].[5] ( ). %6/ %50.[16] 8 197/3±52/6 199/2±35/3.. GLUT4 %22.[12] 4/23 4/32.[17] 8.[16] [21] [3]. B (IR).[8].[18 10]. %3/5...[5]. (TG) 8 193/2±46/4 mg/dl 212/4±76/4..[3] %18/9.[9]

7 [23].[22 23].[22] 8 %9/2..[22] B-100.[22] B-100 %4 B-100 %3/9.[22] B B-100. APO B B [2] B-100..[19]. [9] [22] [16] (%17/3).[3].[21]. LCAT [19]. 106/7±46/5 108/8±31/7 [23] [3] [9] [21].[4]..[22] 4 [23] /3

8 B References 1- Verges B. Lipid modification in type 2 diabetes: the role of and. Fundam Clin Pharmacol Dec; 23(6): Rabbani N, Rabbani N, Chittari MV, Bodmer CW, Zehnder D, Ceriello A, et al. Increased glycation and oxidative damage to apolipoprotein B100 of cholesterol in patients with type 2 diabetes and effect of metformin. Diabetes Apr; 59(4): Zois CE, Tokmakidis SP, Volaklis KA, Kotsa K, Touvra AM, Douda E, et al. Lipoprotein profile, glycemic control and physical fitness after strength and aerobic training in post-menopausal women with type 2 diabetes. Eur J Appl Physiol Aug; 106(6): Iborra RT, Ribeiro IC, Neves MQ, Charf AM, Lottenberg SA, Negrao CE, et al. Aerobic exercise training improves the role of high-density lipoprotein antioxidant and reduces plasma lipid peroxidation in type 2 diabetes mellitus. Scand J Med Sci Sports Dec; 18(6): Agosti V,Graziano S, Artiaco L, Sorrentino G. Biological mechanisms of stroke prevention by physical activity in type 2 diabetes. Acta Neurol Scand Apr; 119(4): Watts GF, Ooi EM, Chan DC. Therapeutic regulation of apob100 metabolism in insulin resistance in vivo. Pharmacol Ther Sep; 123(3): Marwick TH, Hordern MD, Miller T, Chyun DA, Bertoni AG, Blumenthal RS, et al. Exercise training for type 2 diabetes mellitus: impact on cardiovascular risk: a scientific statement from the American Heart Association. Circulation Jun; 119(25): Wang Y, Simar D, Fiatarone Singh M. Adaptations to exercise training within skeletal muscle in adults with type 2 diabetes or impaired glucose tolerance: a systematic review. Diabetes Metab Res Rev Jan; 25(1): Sigal RJ, Kenny GP, Boule NG, Wells GA, Prudhomme D, Fortier M, et al. Effects of aerobic training, resistance training, or both on glycemic control in type 2 diabetes: a randomized trial. Ann Intern Med Sep; 147(6): Sanz C, Gautier JF, Hanaire H. Physical exercise for the prevention and treatment of type 2 diabetes. Diabetes Metab Nov; 36(5): Mogensen M, Vind BF, Hojlund K, Beck-Nielsen H, Sahlin K. Maximal lipid oxidation in patients with type 2 diabetes is normal and shows an adequate increase in response to aerobic training. Diabetes Obes Metab Sep; 11(9): Teixeira-Lemos E, Nunes S, Teixeira F, Reis F. Regular physical exercise training assists in preventing type 2 diabetes development: focus on its antioxidant and anti-inflammatory properties. Cardiovasc Diabetol Jan; 28(10): Osho O, Akinbo S, Osinubi A, Olawale O. Effect of Progressive Aerobic and Resistance Exercises on the Pulmonary functions of Individuals with Type 2 Diabetes in Nigeria. Int J Endocrinol Metab Jan; 10(1):

9 Bweir S, Al-Jarrah M, Almalty AM, Maayah M, Smirnova IV, Novikova L. Resistance exercise training lowers HbA1c more than aerobic training in adults with type 2 diabetes. Diabetol Metab Syndr Dec; 10(1): Robergs RA, Keteyian SJ. Fundamentals of Exercise Physiology for Fitness, Performance, and Health. 2 nd ed. Mc Graw-Hill, 2003: Arora E, Shenoy S, Sandhu JS. Effects of resistance training on metabolic profile of adults with type 2 diabetes. Indian J Med Res May; 129(5): Gordon LA, Morrison EY, McGrowder DA, Young R, Fraser YT, Zamora EM, et al. Effect of exercise therapy on lipid profile and oxidative stress indicators in patients with type 2 diabetes. BMC Complement Altern Med May; 13(8): Cho K, Kim YB. Molecular mechanism of insulin resistance in obesity and type 2 diabetes. Korean J Intern Med Jun; 25(2): Mougios V. Exercise Biochemistry, 1 st ed. Human Kinetics, 2006 Jun; Plotnikoff RC, Courneya K, Trinh L, Karunamuni N, Sigal R. Aerobic physical Activity and resistance training: an application of the theory of planned behavior among Adults with type 2 diabetes in a random, national sample of Canadians. Int J behav Nutri physl Act Dec; (5): Balducci S, Nicolucci D, Fernando E, Cavallo S. Anti-inflammatory effect of exercise training in subjects with type 2 diabetes and the metabolic syndrome is dependent on exercise modalities and independent of weight loss. Nutr Metab Cardiovasc Dis Oct; 20(8): Kawano M, Shono N, Yoshimura T, Yamaguchi M, Hirano T, Hisatomi A. Improved cardiorespiratory fitness correlates with changes in the number and size of small dense : randomized controlled trial with exercise training and dietary instruction. Intern Med Jan; 48(1): Wooten JS, Biggerstaff KD, Ben-Ezra V. Responses of and particle size and distribution to omega-3 fatty acid supplementation and aerobic exercise. J Appl Physiol Sep; 107(3):

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